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Table 1 Analysis of molecular variance (AMOVA) for 17 (non-pseudoautosomal) X-chromosomal markers.

From: Robustness of the inference of human population structure: A comparison of X-chromosomal and autosomal microsatellites

Sample

Number of regions

Number of populations

  

Variance components (%)

  
   

Within populations

Among populations within regions

Among regions

World

1

52

91.1

(89.0, 92.9)

8.9

(7.1, 11.0)

  

World

5

52

89.3

(86.5, 91.8)

4.8

(4.4, 5.3)

5.8

(3.6, 8.5)

World

7

52

90.4

(88.0, 92.5)

4.6

(4.2, 5.1)

4.9

(3.0, 7.3)

World-B97

5

14

85.4

(81.4, 88.7)

7.1

(5.9, 8.2)

7.5

(4.0, 11.8)

Africa

1

6

93.1

(91.2, 94.8)

6.9

(5.2, 8.8)

  

Eurasia

1

21

96.2

(95.4, 96.8)

3.8

(3.2, 4.6)

  

Eurasia

3

21

95.9

(95.1, 96.7)

3.2

(2.7, 3.8)

0.9

(0.4, 1.4)

   Europe

1

8

97.2

(96.3, 98.0)

2.8

(2.0, 3.7)

  

   Middle East

1

4

97.7

(96.9, 98.4)

2.3

(1.6, 3.1)

  

   Central/South Asia

1

9

95.8

(95.0, 96.5)

4.2

(3.5, 5.0)

  

East Asia

1

18

95.3

(94.4, 96.1)

4.7

(3.9, 5.6)

  

Oceania

1

2

91.3

(87.9, 94.6)

8.7

(5.4, 12.1)

  

America

1

5

86.9

(84.7, 88.9)

13.1

(11.1, 15.3)

  
  1. Ninety-five percent confidence intervals (in parentheses) were calculated using 1,000 bootstraps across loci. The World-B97 sample[6, 19] consists of 14 populations that were chosen in order to approximate the sample of Barbujani et al. [10]